There are many conditions that exist in both Pediatric and Adult patients who present to the ED. The author’s group have produced a transient and homolateral facial paralysis in Balb/c mice by inoculating HSV-1 onto the auricle, simulating the symptoms of Bell’s palsy. 2) Furuta Y, Fukuda S, Chida S, et al: Reactivation of herpes simplex virus type 1 in patients with Bell’s palsy. In this study, interferon and IgG administration were performed before and after facial nerve paralysis and continued for 3 days. Histopathologically, severe nerve swelling, inflammatory cell infiltration, and vacuolar degeneration were manifested in the affected facial nerve and nuclei. The HSV-1 DNA in bilateral facial nerve, bilateral brainstem, bilateral trigeminal carrier ganglion, bilateral brain, and blood at different stage was examined with polymerase chain reaction analysis. As with other forms and causes of facial paralysis, symptoms of Bell’s Palsy include facial droop, difficulty with eye closure, inability to move the corners of the mouth.
Nervous system disease: Diseases including Opercular syndrome, Millard-Gubler syndrome can cause facial nerve palsy. This can in some cases lead to cornea ulceration as well. When paralysis is permanent, some advocate facial nerve decompression in selected cases. Prednisolone slightly increases recovery rates if given within 72 hours of onset, whereas no good evidence exists for antivirals. Damage to the nerve which controls facial muscle movement is cause by inflammation. Bell’s palsy is characterized by inflammation of the facial nerve or its nerve sheath. The etiology of DFP and its association with herpes infection should be further clarified.
Hypertension, diabetes, pregnancy, neoplasia, systemic illness and previous ear disease or head injury may be relevant (Adour, 1982). Physical therapy in the form of neuromuscular retraining is also a key component in treatment of Bell’s palsy. However, misdiagnosis of other causes of facial nerve paralysis as Bell’s palsy is not uncommon.